Expanding strategies to resolve psoriasis-like inflammation: Non-canonical NF-κB signaling controls IL-23 production in dendritic cells

Mol Ther. 2024 May 1;32(5):1195-1196. doi: 10.1016/j.ymthe.2024.04.012. Epub 2024 Apr 17.

Authors

Teresa Neuwirth¹, Georg Stary²

Affiliations

¹ Department of Dermatology, Medical University of Vienna, Vienna, Austria; CeMM Research Center for Molecular Medicine, Austrian Academy of Sciences, Vienna, Austria.
² Department of Dermatology, Medical University of Vienna, Vienna, Austria; CeMM Research Center for Molecular Medicine, Austrian Academy of Sciences, Vienna, Austria. Electronic address: georg.stary@meduniwien.ac.at.

PMID: 38631353
PMCID: PMC11081908 (available on 2025-05-01)
DOI: 10.1016/j.ymthe.2024.04.012

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Animals
Dendritic Cells* / immunology
Dendritic Cells* / metabolism
Humans
Inflammation / metabolism
Interleukin-23* / metabolism
NF-kappa B* / metabolism
Psoriasis* / genetics
Psoriasis* / metabolism
Signal Transduction*

Substances

NF-kappa B
Interleukin-23