Rat hair-follicle-associated pluripotent (HAP) stem cells can differentiate into atrial or ventricular cardiomyocytes in culture controlled by specific supplementation

PLoS One. 2024 Jan 26;19(1):e0297443. doi: 10.1371/journal.pone.0297443. eCollection 2024.

Abstract

There has been only limited success to differentiate adult stem cells into cardiomyocyte subtypes. In the present study, we have successfully induced beating atrial and ventricular cardiomyocytes from rat hair-follicle-associated pluripotent (HAP) stem cells, which are adult stem cells located in the bulge area. HAP stem cells differentiated into atrial cardiomyocytes in culture with the combination of isoproterenol, activin A, bone morphogenetic protein 4 (BMP4), basic fibroblast growth factor (bFGF), and cyclosporine A (CSA). HAP stem cells differentiated into ventricular cardiomyocytes in culture with the combination of activin A, BMP4, bFGF, inhibitor of Wnt production-4 (IWP4), and vascular endothelial growth factor (VEGF). Differentiated atrial cardiomyocytes were specifically stained for anti-myosin light chain 2a (MLC2a) antibody. Ventricular cardiomyocytes were specially stained for anti-myosin light chain 2v (MLC2v) antibody. Quantitative Polymerase Chain Reaction (qPCR) showed significant expression of MLC2a in atrial cardiomyocytes and MLC2v in ventricular cardiomyocytes. Both differentiated atrial and ventricular cardiomyocytes showed characteristic waveforms in Ca2+ imaging. Differentiated atrial and ventricular cardiomyocytes formed long myocardial fibers and beat as a functional syncytium, having a structure similar to adult cardiomyocytes. The present results demonstrated that it is possible to induce cardiomyocyte subtypes, atrial and ventricular cardiomyocytes, from HAP stem cells.

MeSH terms

  • Animals
  • Cell Differentiation
  • Dietary Supplements
  • Hair Follicle
  • Myocytes, Cardiac* / metabolism
  • Pluripotent Stem Cells*
  • Rats
  • Vascular Endothelial Growth Factor A / metabolism

Substances

  • Vascular Endothelial Growth Factor A

Grants and funding

This work was partially supported by Grant-in-Aid for Scientific Research (C) 19K08781 from the Ministry of Education, Culture, Sports, Science and Technology of Japan (https://www.mext.go.jp/en/) (to Y.A.), and the Terumo Life Science Foundation (https://www.terumozaidan.or.jp/english/) (to Y.A.) and Parents’ Association Grant of Kitasato University, School of Medicine (to N.T.) and a research grant for young medical doctors and healthcare professionals from SRL, Inc. (to N.T.) and a grant from Kitasato University Graduate School of Medical Sciences (Integrative Research Program for Students 2022) (to N.T.). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.