Modulation of Mitochondrial Function as a Therapeutic Strategy for Neurodegenerative Diseases

J Prev Alzheimers Dis. 2023;10(4):675-685. doi: 10.14283/jpad.2023.108.

Abstract

Despite recent FDA approval of anti-amyloid antibodies for Alzheimer's Disease, strategies that target early molecular mechanisms and could delay or change the disease trajectory are still needed. Mitochondria emerge as a signaling organelle that could modulate multiple molecular mechanisms to enhance cellular bioenergetics and promote neuronal survival. Approaches to enhance mitochondrial function could promote healthy aging delaying the onset of age-related diseases such as Alzheimer's Disease. Some of these strategies have been recently tested in clinical trials. Emerging evidence demonstrates that in response to mild energetic stress, mitochondria could orchestrate a robust adaptive stress response activating multiple neuroprotective mechanism. The objective of this review is to highlight recent development of mitochondria-targeting therapeutics for neurodegenerative diseases, mitochondria complex I inhibitors in particular.

Keywords: Alzheimer’s Disease; Mitochondria; complex I inhibitors; mitochondria targeting therapeutics; mitochondrial signaling; neurodegenerative diseases of aging; neuroprotection.

Publication types

  • Review
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alzheimer Disease*
  • Energy Metabolism
  • Humans
  • Mitochondria / metabolism
  • Neurodegenerative Diseases* / drug therapy