Abstract
The riboflavin analog 10-(4'-chlorophenyl)-3-methylflavin was found to have significant activity against Plasmodium vinckei vinckei when administered orally and parenterally; it was active against P. falciparum in culture. It inhibited mouse erythrocyte glutathione reductase in a dose-dependent manner. When administered orally, 5-deazariboflavin was not active in vivo although it has been shown to have activity against P. falciparum in vitro.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Administration, Oral
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Animals
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Antimalarials / pharmacology
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Antimalarials / therapeutic use*
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Drug Evaluation, Preclinical
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Female
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Injections, Intraperitoneal
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Malaria / drug therapy*
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Mice
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Mice, Inbred CBA
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NADH, NADPH Oxidoreductases / antagonists & inhibitors
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Plasmodium / drug effects*
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Riboflavin / analogs & derivatives*
Substances
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Antimalarials
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NADH, NADPH Oxidoreductases
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Cystine reductase
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Riboflavin