Secretome from Human Mesenchymal Stem Cells-Derived Endothelial Cells Promotes Wound Healing in a Type-2 Diabetes Mouse Model

Int J Mol Sci. 2022 Jan 15;23(2):941. doi: 10.3390/ijms23020941.

Abstract

Tissue regeneration is often impaired in patients with metabolic disorders such as diabetes mellitus and obesity, exhibiting reduced wound repair and limited regeneration capacity. We and others have demonstrated that wound healing under normal metabolic conditions is potentiated by the secretome of human endothelial cell-differentiated mesenchymal stem cells (hMSC-EC). However, it is unknown whether this effect is sustained under hyperglycemic conditions. In this study, the wound healing effect of secretomes from undifferentiated human mesenchymal stem cells (hMSC) and hMSC-EC in a type-2 diabetes mouse model was analyzed. hMSC were isolated from human Wharton's jelly and differentiated into hMSC-EC. hMSC and hMSC-EC secretomes were analyzed and their wound healing capacity in C57Bl/6J mice fed with control (CD) or high fat diet (HFD) was evaluated. Our results showed that hMSC-EC secretome enhanced endothelial cell proliferation and wound healing in vivo when compared with hMSC secretome. Five soluble proteins (angiopoietin-1, angiopoietin-2, Factor de crecimiento fibroblástico, Matrix metallopeptidase 9, and Vascular Endothelial Growth Factor) were enriched in hMSC-EC secretome in comparison to hMSC secretome. Thus, the five recombinant proteins were mixed, and their pro-healing property was evaluated in vitro and in vivo. Functional analysis demonstrated that a cocktail of these proteins enhanced the wound healing process similar to hMSC-EC secretome in HFD mice. Overall, our results show that hMSC-EC secretome or a combination of specific proteins enriched in the hMSC-EC secretome enhanced wound healing process under hyperglycemic conditions.

Keywords: human mesenchymal cells-derived endothelial cells; hyperglycemic conditions; regeneration; secretome.

MeSH terms

  • Angiopoietin-1 / metabolism
  • Angiopoietin-1 / pharmacology
  • Angiopoietin-2 / metabolism
  • Angiopoietin-2 / pharmacology
  • Animals
  • Cell Differentiation
  • Cell Proliferation
  • Cells, Cultured
  • Culture Media, Conditioned / chemistry
  • Culture Media, Conditioned / pharmacology*
  • Diabetes Mellitus, Type 2 / chemically induced
  • Diabetes Mellitus, Type 2 / metabolism*
  • Diet, High-Fat / adverse effects
  • Disease Models, Animal
  • Humans
  • Matrix Metalloproteinase 9 / metabolism
  • Matrix Metalloproteinase 9 / pharmacology
  • Mesenchymal Stem Cells / cytology*
  • Mesenchymal Stem Cells / metabolism
  • Mice
  • Recombinant Proteins / pharmacology*
  • Vascular Endothelial Growth Factor A / metabolism
  • Vascular Endothelial Growth Factor A / pharmacology
  • Wharton Jelly / cytology
  • Wharton Jelly / metabolism
  • Wound Healing / drug effects*

Substances

  • Angiopoietin-1
  • Angiopoietin-2
  • Culture Media, Conditioned
  • Recombinant Proteins
  • Vascular Endothelial Growth Factor A
  • Matrix Metalloproteinase 9