"P2RY8-son" break of tolerance promotes SLE

J Exp Med. 2022 Jan 3;219(1):e20211972. doi: 10.1084/jem.20211972. Epub 2021 Dec 13.

Abstract

In this issue of JEM, He et al. (2021. J. Exp. Med.https://doi.org/10.1084/jem.20211004) associate novel P2RY8 genetic variants to lupus, expanding the field of monogenic autoimmunity. The authors demonstrate that P2RY8 prevents the expansion of DNA-reactive B cells by restraining B cell mobility and activation within the germinal center.

Publication types

  • Comment

MeSH terms

  • B-Lymphocytes
  • DNA
  • Germinal Center*
  • Humans
  • Lupus Erythematosus, Systemic* / genetics
  • Nuclear Family
  • Receptors, Purinergic P2Y

Substances

  • P2RY8 protein, human
  • Receptors, Purinergic P2Y
  • DNA