Impaired kinetic properties of hypoxanthine-guanine phosphoribosyl transferase as a cause of uric acid nephropathy in early infancy

Eur J Pediatr. 1987 Nov;146(6):595-7. doi: 10.1007/BF02467362.

Abstract

A 6-month-old infant presented with failure to thrive, hyperuricaemia and renal insufficiency. The hyperuricaemia was due to uric acid over-production. The level of hypoxanthine-guanine phosphoribosyltransferase (HGPRT) activity was found to be normal. However, a two-fold increase in the Km of the enzyme to hypoxanthine as well as in the Vmax values was observed. It seems therefore, that in cases of uric acid over-production, screening tests of HGPRT activity may be insufficient and additional kinetic properties of the enzyme should be tested.

Publication types

  • Case Reports

MeSH terms

  • Acute Kidney Injury / enzymology
  • Acute Kidney Injury / etiology*
  • Consanguinity
  • Failure to Thrive / enzymology
  • Failure to Thrive / etiology
  • Humans
  • Hypoxanthine Phosphoribosyltransferase / metabolism*
  • Infant
  • Kinetics
  • Male
  • Uric Acid / metabolism*

Substances

  • Uric Acid
  • Hypoxanthine Phosphoribosyltransferase