Enhancement of antibody-dependent cellular cytotoxicity is associated with treatment response to extracorporeal photopheresis in Sézary syndrome

Oncoimmunology. 2021 Jan 31;10(1):1873530. doi: 10.1080/2162402X.2021.1873530.

Abstract

Sézary syndrome (SS) is a rare, leukemic type of cutaneous T-cell lymphoma (CTCL), for which extracorporeal photopheresis (ECP) is a first-line therapy. Reliable biomarkers to objectively monitor the response to ECP in patients with SS are missing. We examined the quantitative and qualitative impact of ECP on natural killer (NK) cell activity in SS patients, and especially their functional ability for antibody-dependent cell-mediated cytotoxicity (ADCC). Further, we addressed the question whether the magnitude of the effect on ADCC can be associated with the anti-cancer efficacy of ECP in SS patients. We assessed numbers of NK cells, ADCC activity, and treatment response based on blood tumor staging in a cohort of 13 SS patients (8 women, 5 men) treated with ECP as a first-line therapy. Blood samples were collected before treatment start and after an average of 9 months of uninterrupted ECP treatment. NK cell numbers were reduced in SS patients compared to healthy individuals and showed a tendency of recovery after long-term ECP treatment, independent of the clinical response to treatment. Patients with marginal increase (≤1.5 AU-fold) or lack of increase in ADCC activity failed to respond clinically to treatment, while patients with an increased ADCC activity showed a reduction in blood tumor burden. NK-mediated ADCC is selectively enhanced and might be a mechanism underlying the effect of ECP while in addition it can possibly serve as a reliable biomarker to objectively monitor response to ECP in patients with SS.

Keywords: Cutaneous T cell lymphoma; Sézary syndrome; biomarker; cellular cytotoxicity; dermatology; extracorporeal photophoresis; mycosis fungoides; treatment response.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antibody-Dependent Cell Cytotoxicity
  • Female
  • Humans
  • Lymphoma, T-Cell, Cutaneous* / pathology
  • Male
  • Neoplasm Staging
  • Photopheresis*
  • Skin Neoplasms* / therapy

Grants and funding

This work was supported by the European Molecular Biology Organization [7637]; Schweizerischer Nationalfonds zur Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung [PMPDP3_151326]; Swiss Cancer Research Foundation KFS-4243-08-2017]; Polish National Science Centre [2015/19/B/NZ6/02862]; Forschungskredit of the University of Zurich [FK-15-040]; anonymous Foundation; European Academy of Dermatology and Venereology [PPRC-2019-20]; Jubiläumsstiftung von SwissLife; European Commission Horizon 2020 Programme [692180-STREAMH2020-TWINN-2015]; Promedica Stiftung [1406/M and 1412/M]; Clinical Research Priority Program (CRPP) of the University of Zurich as a funder.