Elevated Circulating IL-10 Producing Breg, but Not Regulatory B Cell Levels, Restrain Antibody-Mediated Rejection After Kidney Transplantation

Front Immunol. 2021 Jan 28:11:627496. doi: 10.3389/fimmu.2020.627496. eCollection 2020.

Abstract

Background: Antibody-mediated rejection (AMR) occupies a major position for chronic rejection after kidney transplantation. Regulatory B cell (Breg) has been reported to have an inhibitory immune function, which contributes to the resistance for AMR.

Methods: A nested case-control study for nine healthy donors, 25 stable (ST) patients, and 18 AMR patients was performed to determine the type of Breg in maintaining immune tolerance and preventing AMR.

Results: Compared to the ST group, circulating interleukin (IL)-10+ Bregs, but not Bregs, significantly decreased. The receiver operating characteristic (ROC) curve analysis revealed that rather than the circulating Bregs, decreased circulating IL-10+ Breg levels were positively associated with AMR. However, kidney B cell and IL-10 infiltration was significantly increased in the AMR group with high expression of C-X-C motif chemokine 13 (CXCL13). In addition, circulating IL-10+ Bregs, rather than Bregs, remained higher than those at pre-operation, during the 90-day post-operation in immune homeostasis.

Conclusion: The circulating IL-10+ Breg levels are more appropriate measures for assessing the resistance of AMR after kidney transplantation.

Keywords: Breg phenotyping; antibody-mediated rejection; dynamic; homeostasis; kidney transplantation.

Publication types

  • Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • B-Lymphocytes, Regulatory / immunology*
  • B-Lymphocytes, Regulatory / pathology
  • Female
  • Graft Rejection / immunology*
  • Graft Rejection / pathology
  • Humans
  • Interleukin-10 / immunology*
  • Isoantibodies / immunology*
  • Kidney / immunology*
  • Kidney / pathology
  • Kidney Transplantation*
  • Male
  • Middle Aged

Substances

  • IL10 protein, human
  • Isoantibodies
  • Interleukin-10