Abstract
Recent studies have highlighted a major role for cancer-associated fibroblasts (CAFs) in promoting immunotherapy resistance by excluding T cells from tumours. Recently, we showed that CAFs can be effectively targeted by inhibiting the enzyme NOX4; this 'normalises' CAFs and overcomes immunotherapy resistance. Here we discuss our study and other strategies for CAF targeting.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antineoplastic Agents, Immunological / therapeutic use*
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Antineoplastic Combined Chemotherapy Protocols / therapeutic use
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Cancer-Associated Fibroblasts / drug effects*
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Cancer-Associated Fibroblasts / pathology
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Cancer-Associated Fibroblasts / physiology
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Drug Resistance, Neoplasm* / drug effects
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Drug Synergism
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Humans
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Immunotherapy
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Molecular Targeted Therapy / methods*
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Molecular Targeted Therapy / trends
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NADPH Oxidase 4 / antagonists & inhibitors
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Neoplasms / pathology
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Neoplasms / therapy
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Pyrazolones / administration & dosage
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Pyrazolones / pharmacology*
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Pyridones / administration & dosage
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Pyridones / pharmacology*
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T-Lymphocytes / drug effects
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T-Lymphocytes / immunology
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T-Lymphocytes / pathology*
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Tumor Microenvironment / drug effects
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Tumor Microenvironment / immunology
Substances
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Antineoplastic Agents, Immunological
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Pyrazolones
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Pyridones
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setanaxib
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NADPH Oxidase 4
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NOX4 protein, human