Dynamically Expressed miR-BART16 Functions as a Suppressor of CAND1 in Infectious Mononucleosis Caused by Epstein-Barr Virus in Children

Ann Clin Lab Sci. 2020 May;50(3):371-377.

Abstract

Objective: MiR-BART16 is a newly discovered Epstein-Barr Virus-encoded microRNA (miRNA). We aimed to explore the role of EBV-miR-BART16 in infectious mononucleosis (IM).

Methods: Peripheral blood lymphocyte subsets were analyzed in 30 IM and 10 healthy children by flow cytometry. MiR-BART16 and its targets were measured by real-time PCR, western blot, ELISA, and dual-luciferase assay.

Results: Serum miR-BART16 expression was significantly higher in the IM children than that in the healthy children, and was positively correlated with EBV copy number. Receiver operating characteristic analysis revealed serum miR-BART16 could differentiate IM and healthy individuals (P=0.0041). CAND1 was targeted and downregulated by miR-BART16 in an EBV infection-dependent way.

Conclusions: These results highlight that EBV-miR-BART16 plays an important role in regulating the expression of CAND1 to affect pediatric IM.

Keywords: CAND1; Epstein-Barr virus; Infectious mononucleosis; miRNA.

MeSH terms

  • Child
  • Epstein-Barr Virus Infections / blood
  • Epstein-Barr Virus Infections / virology
  • Female
  • Flow Cytometry / methods
  • Gene Expression / genetics
  • Gene Expression Regulation / genetics
  • Herpesvirus 4, Human / genetics*
  • Herpesvirus 4, Human / pathogenicity
  • Humans
  • Infectious Mononucleosis / genetics*
  • Infectious Mononucleosis / metabolism
  • Lymphocyte Subsets
  • Male
  • MicroRNAs / genetics*
  • RNA, Viral / genetics
  • Real-Time Polymerase Chain Reaction
  • Transcription Factors / genetics
  • Transcription Factors / metabolism

Substances

  • CAND1 protein, human
  • MicroRNAs
  • RNA, Viral
  • Transcription Factors