Both male and female obese ZSF1 rats develop cardiac dysfunction in obesity-induced heart failure with preserved ejection fraction

PLoS One. 2020 May 6;15(5):e0232399. doi: 10.1371/journal.pone.0232399. eCollection 2020.

Abstract

Heart failure with a preserved ejection fraction (HFpEF) is associated with multiple comorbidities, such as old age, hypertension, type 2 diabetes and obesity and is more prevalent in females. Although the male obese ZSF1 rat has been proposed as a suitable model to study the development of diastolic dysfunction and early HFpEF, studies in female animals have not been performed yet. Therefore, we aimed to characterize the cardiac phenotype in female obese ZSF1 rats and their lean counterparts. Additionally, we aimed to investigate whether differences exist in disease progression in obese male and female ZSF1 rats. Therefore, male and female ZSF1 rats, lean as well as obese (N = 6-9/subgroup), were used. Every two weeks, from 12 to 26 weeks of age, systolic blood pressure and echocardiographic measurements were performed, and venous blood was sampled. Female obese ZSF1 rats, as compared to female lean ZSF1 rats, developed diastolic dysfunction with cardiac hypertrophy and fibrosis in the presence of severe dyslipidemia, increased plasma growth differentiation factor 15 and mild hypertension, and preservation of systolic function. Although obese female ZSF1 rats did not develop hyperglycemia, their diastolic dysfunction was as severe as in the obese males. Taken together, the results from the present study suggest that the female obese ZSF1 rat is a relevant animal model for HFpEF with multiple comorbidities, suitable for investigating novel therapeutic interventions.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blood Pressure / physiology
  • Collagen / metabolism
  • Diastole / physiology
  • Disease Models, Animal
  • Echocardiography
  • Female
  • Heart Failure / diagnostic imaging
  • Heart Failure / etiology*
  • Heart Failure / physiopathology
  • Male
  • Metabolic Syndrome / complications
  • Myocardium / metabolism
  • Myocardium / pathology
  • Obesity / complications*
  • Rats
  • Rats, Inbred SHR
  • Rats, Zucker
  • Sex Characteristics
  • Stroke Volume / physiology
  • Thinness / physiopathology
  • Ventricular Remodeling / physiology

Substances

  • Collagen

Grants and funding

This study was supported by a grant from the Netherlands CardioVascular Research Initiative: an initiative with support of the Dutch Heart Foundation [CVON2014-11 (RECONNECT)] and by the German Center for Cardiovascular Research (DZHK; 81Z0600207).The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.