Prospective study reveals a microbiome signature that predicts the occurrence of post-operative enterocolitis in Hirschsprung disease (HSCR) patients

Gut Microbes. 2020 Jul 3;11(4):842-854. doi: 10.1080/19490976.2020.1711685. Epub 2020 Jan 16.

Abstract

Hirschsprung disease (HSCR) is a birth defect with an approximate incidence of 1/5,000 live births, and up to one-third of HSCR patients develop Hirschsprung-associated enterocolitis (HAEC), the leading cause of HSCR-related death. Very little is known about the pathogenesis, prevention, and early diagnosis of HAEC. Here, we used a prospective study to investigate the enteric microbiome composition at the time of surgery as a predictor for developing postoperative HAEC. We identified a microbiome signature containing 21 operational taxonomic units (OTUs) that can potentially predict postoperative HAEC with ~85% accuracy. Furthermore, we identified exclusive breastfeeding as a novel protective factor for total HAEC (i.e., preoperative and postoperative HAEC combined). In addition, we discovered that breastfeeding was associated with a lowered risk for HAEC potentially mediated by modulating the gut microbiome composition characterized by a lower abundance of Gram-negative bacteria and lower LPS concentrations. In conclusion, modulating the gut microbiome by encouraging breastfeeding might prevent HAEC progression in HSCR patients.

Keywords: Hirschsprung disease; Hirschsprung-associated enterocolitis; exclusive breastfeeding; the enteric microbiome.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bacteria / classification
  • Bacteria / growth & development*
  • Breast Feeding
  • Case-Control Studies
  • Colon / microbiology
  • Enterocolitis / etiology*
  • Female
  • Gastrointestinal Microbiome*
  • Hirschsprung Disease / complications
  • Hirschsprung Disease / surgery*
  • Humans
  • Infant
  • Intestinal Mucosa / microbiology*
  • Lipopolysaccharides / metabolism
  • Male
  • Postoperative Complications*
  • Prospective Studies

Substances

  • Lipopolysaccharides

Grants and funding

This work was supported by the Jiangsu Provincial Key Research and Development Program [BE2017609];National Natural Science Foundation of China [NSFC 81570467];Nanjing Medical Science and Technique Development Foundation [ZKX17039];Priority Academic Program Development of Jiangsu Higher Education Institutions [PAPD].