Steric constraints control processing of glycosylphosphatidylinositol anchors in Trypanosoma brucei

J Biol Chem. 2020 Feb 21;295(8):2227-2238. doi: 10.1074/jbc.RA119.010847. Epub 2020 Jan 13.

Abstract

The transferrin receptor (TfR) of the bloodstream form (BSF) of Trypanosoma brucei is a heterodimer comprising glycosylphosphatidylinositol (GPI)-anchored expression site-associated gene 6 (ESAG6 or E6) and soluble ESAG7. Mature E6 has five N-glycans, consisting of three oligomannose and two unprocessed paucimannose structures. Its GPI anchor is modified by the addition of 4-6 α-galactose residues. TfR binds tomato lectin (TL), specific for N-acetyllactosamine (LacNAc) repeats, and previous studies have shown transport-dependent increases in E6 size consistent with post-glycan processing in the endoplasmic reticulum. Using pulse-chase radiolabeling, peptide-N-glycosidase F treatment, lectin pulldowns, and exoglycosidase treatment, we have now investigated TfR N-glycan and GPI processing. E6 increased ∼5 kDa during maturation, becoming reactive with both TL and Erythrina cristagalli lectin (ECL, terminal LacNAc), indicating synthesis of poly-LacNAc on paucimannose N-glycans. This processing was lost after exoglycosidase treatment and after RNAi-based silencing of TbSTT3A, the oligosaccharyltransferase that transfers paucimannose structures to nascent secretory polypeptides. These results contradict previous structural studies. Minor GPI processing was also observed, consistent with α-galactose addition. However, increasing the spacing between E6 protein and the GPI ω-site (aa 4-7) resulted in extensive post-translational processing of the GPI anchor to a form that was TL/ECL-reactive, suggesting the addition of LacNAc structures, confirmed by identical assays with BiPNHP, a non-N-glycosylated GPI-anchored reporter. We conclude that BSF trypanosomes can modify GPIs by generating structures reminiscent of those present in insect-stage trypanosomes and that steric constraints, not stage-specific expression of glycosyltransferases, regulate GPI processing.

Keywords: N-glycan processing; N-linked glycosylation; glycobiology; glycosylphosphatidylinositol (GPI anchor); glycosylphosphatidylinositol processing; kinetoplastid protozoa; transferrin; transferrin receptor; trypanosome.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Glycosides / metabolism
  • Glycosylation
  • Glycosylphosphatidylinositols / chemistry*
  • Glycosylphosphatidylinositols / metabolism*
  • Lectins / metabolism
  • Polysaccharides / chemistry
  • Polysaccharides / metabolism
  • Protozoan Proteins / metabolism
  • Receptors, Transferrin / metabolism
  • Substrate Specificity
  • Trypanosoma brucei brucei / metabolism*

Substances

  • Glycosides
  • Glycosylphosphatidylinositols
  • Lectins
  • Polysaccharides
  • Protozoan Proteins
  • Receptors, Transferrin