Skeletal muscle mitochondrial beta-oxidation. A study of the products of oxidation of [U-14C]hexadecanoate by h.p.l.c. using continuous on-line radiochemical detection

Biochem J. 1988 Jul 15;253(2):541-7. doi: 10.1042/bj2530541.

Abstract

Well-coupled mitochondrial fractions were prepared from rat skeletal muscle without the use of proteolytic enzymes. The products of [U-14C]hexadecanoate oxidation by rat skeletal muscle mitochondrial fractions were analysed by h.p.l.c. with on-line radiochemical detection. In the presence of 1 mM-carnitine, 70% of the products is acetylcarnitine. In agreement with Veerkamp et al. [Veerkamp, van Moerkerk, Glatz, Zuurveld, Jacobs & Wagenmakers (1986) Biochem. Med. Metab. Biol. 35, 248-259] 14CO2 release is shown to be an unreliable estimate of flux through beta-oxidation in skeletal muscle mitochondrial fractions. The flux through beta-oxidation is recorded unambiguously polarographically in the presence of 1 mM-carnitine and the absence of citrate cycle intermediates.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcarnitine / metabolism
  • Animals
  • Carbon Radioisotopes
  • Carnitine / pharmacology
  • Chromatography, High Pressure Liquid
  • Malates / pharmacology
  • Male
  • Mitochondria, Muscle / drug effects
  • Mitochondria, Muscle / metabolism*
  • Oxidation-Reduction
  • Palmitic Acids / metabolism*
  • Palmitoylcarnitine / metabolism
  • Rats
  • Rats, Inbred Strains
  • Tissue Distribution

Substances

  • Carbon Radioisotopes
  • Malates
  • Palmitic Acids
  • Palmitoylcarnitine
  • Acetylcarnitine
  • malic acid
  • Carnitine