Leptin-induced Trafficking of KATP Channels: A Mechanism to Regulate Pancreatic β-cell Excitability and Insulin Secretion

Int J Mol Sci. 2019 May 30;20(11):2660. doi: 10.3390/ijms20112660.

Abstract

The adipocyte hormone leptin was first recognized for its actions in the central nervous system to regulate energy homeostasis but has since been shown to have direct actions on peripheral tissues. In pancreatic β-cells leptin suppresses insulin secretion by increasing KATP channel conductance, which causes membrane hyperpolarization and renders β-cells electrically silent. However, the mechanism by which leptin increases KATP channel conductance had remained unresolved for many years following the initial observation. Recent studies have revealed that leptin increases surface abundance of KATP channels by promoting channel trafficking to the β-cell membrane. Thus, KATP channel trafficking regulation has emerged as a mechanism by which leptin increases KATP channel conductance to regulate β-cell electrical activity and insulin secretion. This review will discuss the leptin signaling pathway that underlies KATP channel trafficking regulation in β-cells.

Keywords: ATP-sensitive potassium (KATP) channel; Insulin secretion; Leptin; β-cell.

Publication types

  • Review

MeSH terms

  • Animals
  • Biomarkers
  • Glucose / metabolism
  • Humans
  • Insulin Secretion*
  • Insulin-Secreting Cells / metabolism*
  • KATP Channels / metabolism*
  • Leptin / metabolism*
  • Protein Binding
  • Protein Transport
  • Signal Transduction

Substances

  • Biomarkers
  • KATP Channels
  • Leptin
  • Glucose