Tks5 and Dynamin-2 enhance actin bundle rigidity in invadosomes to promote myoblast fusion

J Cell Biol. 2019 May 6;218(5):1670-1685. doi: 10.1083/jcb.201809161. Epub 2019 Mar 20.

Abstract

Skeletal muscle development requires the cell-cell fusion of differentiated myoblasts to form muscle fibers. The actin cytoskeleton is known to be the main driving force for myoblast fusion; however, how actin is organized to direct intercellular fusion remains unclear. Here we show that an actin- and dynamin-2-enriched protrusive structure, the invadosome, is required for the fusion process of myogenesis. Upon differentiation, myoblasts acquire the ability to form invadosomes through isoform switching of a critical invadosome scaffold protein, Tks5. Tks5 directly interacts with and recruits dynamin-2 to the invadosome and regulates its assembly around actin filaments to strengthen the stiffness of dynamin-actin bundles and invadosomes. These findings provide a mechanistic framework for the acquisition of myogenic fusion machinery during myogenesis and reveal a novel structural function for Tks5 and dynamin-2 in organizing actin filaments in the invadosome to drive membrane fusion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actin Cytoskeleton / physiology*
  • Animals
  • Cell Communication
  • Cell Differentiation
  • Cell Fusion*
  • Cell Movement
  • Cells, Cultured
  • Dynamin II / metabolism*
  • Membrane Fusion*
  • Mice
  • Myoblasts / cytology
  • Myoblasts / physiology*
  • Phosphate-Binding Proteins / metabolism*
  • Podosomes / metabolism*

Substances

  • Fish protein, mouse
  • Phosphate-Binding Proteins
  • DNM2 protein, mouse
  • Dynamin II