Targeting PAK1 with the Small Molecule Drug AK963/40708899 Suppresses Gastric Cancer Cell Proliferation and Invasion by Downregulation of PAK1 Activity and PAK1-Related Signaling Pathways

Anat Rec (Hoboken). 2019 Sep;302(9):1571-1579. doi: 10.1002/ar.24095. Epub 2019 Mar 27.

Abstract

PAK1 (p21-activated kinase 1) is a serine/threonine protein kinase which has been initially identified as downstream effector of the Rho GTPase family. In previous research, PAK1 has been involved in the regulation of diverse cellular processes, such as cell motility, cell proliferation, gene transcription, cytoskeletal rearrangement, and cell invasion. Hyper-activation of PAK1 was constantly observed in a variety of human cancer which make it a potential target of novel anti-tumor drugs. To date, a great number of attentions focus on identifying the PAK1 inhibitors in medical and pharmaceutical fields. In this article, we found that a novel and potent PAK1 inhibitor, AK963/40708899, suppressed the proliferation of human gastric cancer cells significantly by downregulation of PAK1-NF-κB-cyclinB1 pathway. In addition, AK963/40708899 inhibited the formation of filopodia and promoted cell adhesion which in turn inhibited invasive potential of gastric cells by negatively regulating PAK1-LIMKl-cofilin and PAK1-ERK-FAK pathways. Considering our result, AK963/40708899 would be a possible candidate for PAK1 targeted anti-tumor drug. Anat Rec, 302:1571-1579, 2019. © 2019 American Association for Anatomy.

Keywords: AK963/40708899; PAK1; gastric cancer; inhibitor.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Apoptosis
  • Biomarkers, Tumor
  • Cell Cycle
  • Cell Proliferation*
  • Cyclin B1 / genetics
  • Cyclin B1 / metabolism
  • Gene Expression Regulation, Neoplastic / drug effects*
  • Humans
  • MAP Kinase Signaling System / drug effects*
  • NF-kappa B / genetics
  • NF-kappa B / metabolism
  • Neoplasm Invasiveness
  • Small Molecule Libraries / pharmacology*
  • Stomach Neoplasms / drug therapy*
  • Stomach Neoplasms / metabolism
  • Stomach Neoplasms / pathology
  • Tumor Cells, Cultured
  • Xenograft Model Antitumor Assays
  • p21-Activated Kinases / antagonists & inhibitors*
  • p21-Activated Kinases / metabolism

Substances

  • Antineoplastic Agents
  • Biomarkers, Tumor
  • CCNB1 protein, human
  • Cyclin B1
  • NF-kappa B
  • Small Molecule Libraries
  • PAK1 protein, human
  • p21-Activated Kinases