Discovery of Myricetin as a Potent Inhibitor of Human Flap Endonuclease 1, Which Potentially Can Be Used as Sensitizing Agent against HT-29 Human Colon Cancer Cells

J Agric Food Chem. 2019 Feb 13;67(6):1656-1665. doi: 10.1021/acs.jafc.8b05447. Epub 2019 Jan 29.

Abstract

Human flap endonuclease 1 (hFEN1) is instrumental in DNA replication and repair. It is able to cleave the 5' single-stranded protrusion (also known as 5' flap) resulting from strand displacement reactions. In light of its crucial functions, hFEN1 is now deemed as a nontrivial target in the DNA damage response system for anticancer drug development. Herein, we report that myricetin and some natural flavonoids are able to inhibit hFEN1. Structure-activity relationship, inhibitory mechanisms, molecular docking, and cancer cell-based assays have been performed. Our original findings expand the activity of flavonoids and may pave the way for flavonoid-assisted targeted cancer therapy.

Keywords: colon cancer treatment; flavonoids; human flap endonuclease 1 (hFEN1); inhibitor; molecular docking; myricetin; sensitizing effect; structure−activity relationship analysis.

MeSH terms

  • Colonic Neoplasms / drug therapy
  • Colonic Neoplasms / enzymology*
  • Colonic Neoplasms / genetics
  • Enzyme Inhibitors / chemistry*
  • Enzyme Inhibitors / pharmacology
  • Flap Endonucleases / antagonists & inhibitors*
  • Flap Endonucleases / genetics
  • Flap Endonucleases / metabolism
  • Flavonoids / chemistry*
  • Flavonoids / pharmacology
  • HT29 Cells
  • Humans
  • Molecular Docking Simulation

Substances

  • Enzyme Inhibitors
  • Flavonoids
  • myricetin
  • Flap Endonucleases
  • FEN1 protein, human