Bmi1 marks gastric stem cells located in the isthmus in mice

J Pathol. 2019 Jun;248(2):179-190. doi: 10.1002/path.5244. Epub 2019 Feb 20.

Abstract

In the mammalian stomach, the isthmus has been considered as a stem cell zone. However, various locations and proliferative activities of gastric stem cells have been reported. We focused here on the stem cell marker Bmi1, a polycomb group protein, aiming to elucidate the characteristics of Bmi1-expressing cells in the stomach and to examine their stem cell potential. We investigated the Bmi1-expressing cell lineage in Bmi1-CreERT; Rosa26-YFP, LacZ or Rosa26-Confetti mice. We examined the in vivo and ex vivo effects of Bmi1-expressing cell ablation by using Bmi1-CreERT; Rosa26-iDTR mice. The Bmi1 lineage was also traced during regeneration after high-dose tamoxifen-, irradiation- and acetic acid-induced mucosal injuries. In the lineage-tracing experiments using low-dose tamoxifen, Bmi1-expressing cells in the isthmus of the gastric antrum and corpus provided progeny bidirectionally, towards both the luminal and basal sides over 6 months. In gastric organoids, Bmi1-expressing cells also provided progeny. Ablation of Bmi1-expressing cells resulted in impaired gastric epithelium in both mouse stomach and organoids. After high-dose tamoxifen-induced gastric mucosal injury, Bmi1-expressing cell lineages expanded and fully occupied all gastric glands of the antrum and the corpus within 7 days after tamoxifen injection. After irradiation- and acetic acid-induced gastric mucosal injuries, Bmi1-expressing cells also contributed to regeneration. In conclusion, Bmi1 is a gastric stem cell marker expressed in the isthmus of the antrum and corpus. Bmi1-expressing cells have stem cell potentials, both under physiological conditions and during regeneration after gastric mucosal injuries. Copyright © 2019 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.

Keywords: gastric ulcer; homeostasis; radiation injury; regeneration.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetic Acid
  • Animals
  • Biomarkers / metabolism
  • Cell Differentiation
  • Cell Lineage
  • Cell Proliferation
  • Disease Models, Animal
  • Fluorouracil / toxicity
  • Gene Expression Regulation, Developmental
  • Humans
  • Mice, Transgenic
  • Polycomb Repressive Complex 1 / genetics
  • Polycomb Repressive Complex 1 / metabolism*
  • Proto-Oncogene Proteins / genetics
  • Proto-Oncogene Proteins / metabolism*
  • Pyloric Antrum / drug effects
  • Pyloric Antrum / embryology
  • Pyloric Antrum / metabolism*
  • Pyloric Antrum / radiation effects
  • Regeneration
  • Signal Transduction
  • Stem Cells / drug effects
  • Stem Cells / metabolism*
  • Stem Cells / radiation effects
  • Stomach Ulcer / chemically induced
  • Stomach Ulcer / metabolism
  • Stomach Ulcer / pathology
  • Tamoxifen / toxicity

Substances

  • BMI1 protein, human
  • Biomarkers
  • Bmi1 protein, mouse
  • Proto-Oncogene Proteins
  • Tamoxifen
  • Polycomb Repressive Complex 1
  • Acetic Acid
  • Fluorouracil