New Insights Into the Biology of CD8 Regulatory T Cells

Adv Immunol. 2018:140:1-20. doi: 10.1016/bs.ai.2018.09.001. Epub 2018 Oct 9.

Abstract

Regulatory T cells are central mediators of immune regulation and play an essential role in the maintenance of immune homeostasis in the steady state and under pathophysiological conditions. Disruption of CD8 Treg-dependent recognition of Qa-1-restricted self-antigens can result in dysregulated immune responses, tissue damage, autoimmune disease and cancer. Recent progress in studies on regulatory T cells of the CD8 lineage has provided new biological insight into this specialized regulatory T cell subpopulation. Identification of the Helios transcription factor as an essential control element for the differentiation and function of CD8 regulatory T cells has led to a better understanding of the unique genetic program of these cells. Recent analyses of T-cell receptor usage and antigen recognition by Qa-1-restricted CD8 Treg have provided additional insight into the unusual biological function of this regulatory CD8 lineage. Here we summarize recent advances in our understanding of CD8 regulatory T cells with emphasis on lineage commitment, differentiation and stability.

Keywords: Helios; Inflammation; Qa-1; Suppression; Treg lineage commitment; Treg lineage stability; Tumor microenvironment.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Review

MeSH terms

  • Animals
  • Autoantigens
  • Autoimmune Diseases / immunology*
  • CD8 Antigens / metabolism
  • Cell Differentiation
  • Cell Movement
  • Gene Expression Regulation
  • Histocompatibility Antigens Class I / metabolism
  • Homeostasis
  • Humans
  • Ikaros Transcription Factor / genetics
  • Lymphocyte Activation
  • Neoplasms / immunology*
  • Receptors, Antigen, T-Cell / metabolism
  • T-Lymphocytes, Regulatory / immunology*

Substances

  • Autoantigens
  • CD8 Antigens
  • Histocompatibility Antigens Class I
  • IKZF2 protein, human
  • Q surface antigens
  • Receptors, Antigen, T-Cell
  • Ikaros Transcription Factor