Identifying the genetic causes for prenatally diagnosed structural congenital anomalies (SCAs) by whole-exome sequencing (WES)

BMC Med Genomics. 2018 Oct 25;11(1):93. doi: 10.1186/s12920-018-0409-z.

Abstract

Background: Whole-exome sequencing (WES) has become an invaluable tool for genetic diagnosis in paediatrics. However, it has not been widely adopted in the prenatal setting. This study evaluated the use of WES in prenatal genetic diagnosis in fetuses with structural congenital anomalies (SCAs) detected on prenatal ultrasound.

Method: Thirty-three families with fetal SCAs on prenatal ultrasonography and normal chromosomal microarray results were recruited. Genomic DNA was extracted from various fetal samples including amniotic fluid, chorionic villi, and placental tissue. Parental DNA was extracted from peripheral blood when available. We used WES to sequence the coding regions of parental-fetal trios and to identify the causal variants based on the ultrasonographic features of the fetus.

Results: Pathogenic mutations were identified in three families (n = 3/33, 9.1%), including mutations in DNAH11, RAF1 and CHD7, which were associated with primary ciliary dyskinesia, Noonan syndrome, and CHARGE syndrome, respectively. In addition, variants of unknown significance (VUSs) were detected in six families (18.2%), in which genetic changes only partly explained prenatal features.

Conclusion: WES identified pathogenic mutations in 9.1% of fetuses with SCAs and normal chromosomal microarray results. Databases for fetal genotype-phenotype correlations and standardized guidelines for variant interpretation in prenatal diagnosis need to be established to facilitate the use of WES for routine testing in prenatal diagnosis.

Keywords: Phenotyping; Prenatal exome; Variants of unknown clinical significance.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amniotic Fluid / metabolism
  • Axonemal Dyneins / genetics
  • CHARGE Syndrome / diagnosis
  • CHARGE Syndrome / genetics*
  • Ciliary Motility Disorders / diagnosis
  • Ciliary Motility Disorders / genetics*
  • DNA / isolation & purification
  • DNA / metabolism
  • DNA Helicases / genetics
  • DNA-Binding Proteins / genetics
  • Exome Sequencing*
  • Female
  • Fetus / metabolism
  • Humans
  • Noonan Syndrome / diagnosis
  • Noonan Syndrome / genetics*
  • Phenotype
  • Placenta / metabolism
  • Pregnancy
  • Prenatal Diagnosis
  • Proto-Oncogene Proteins c-raf / genetics
  • Ultrasonography, Prenatal

Substances

  • DNA-Binding Proteins
  • DNA
  • Proto-Oncogene Proteins c-raf
  • Raf1 protein, human
  • DNA Helicases
  • CHD7 protein, human
  • Axonemal Dyneins
  • DNAH11 protein, human