DT-Diaphorase Prevents Aminochrome-Induced Lysosome Dysfunction in SH-SY5Y Cells

Neurotox Res. 2019 Jan;35(1):255-259. doi: 10.1007/s12640-018-9953-8. Epub 2018 Sep 10.

Abstract

Aminochrome has been reported to induce lysosomal dysfunction by inhibiting the vacuolar H-type ATPase localized in lysosome membrane. DT-diaphorase has been proposed to prevent aminochrome neurotoxicity but it is unknown whether this enzyme prevents aminochrome-induced lysosomal dysfunction. In the present study, we tested the protective role of DT-diaphorase in lysosomal dysfunction by generating a cell line (SH-SY5YsiNQ7) with a stable expression of a siRNA against DT-diaphorase with only 10% expression of mRNA enzyme. The cells differentiated with retinoic acid and 12-o-tetradecanoylphorbol-13-acetate show a significant increase in the expression of tyrosine hydroxylase, vesicular monoamine transporter-2, and dopamine transporter. The incubation of SH-SY5YsiNQ7 cells with 10 μM aminochrome resulted in a significant decrease of lysosome pH determined by using acridine orange, while aminochrome has no effect on SH-SY5Y cells. These results support the proposed protective role of DT-diaphorase against aminochrome-induced lysosomal dysfunction.

Keywords: Aminochrome; DT-Diaphorase; Dopamine; Lysosomal dysfunction; Neuroprotection; Neurotoxicity.

MeSH terms

  • Animals
  • Cell Line, Tumor
  • Hydrogen-Ion Concentration
  • Indolequinones / toxicity*
  • Lysosomes / drug effects*
  • Lysosomes / enzymology*
  • Mice
  • NAD(P)H Dehydrogenase (Quinone) / antagonists & inhibitors
  • NAD(P)H Dehydrogenase (Quinone) / genetics
  • NAD(P)H Dehydrogenase (Quinone) / metabolism*
  • Neuroprotection / physiology*

Substances

  • Indolequinones
  • aminochrome 1
  • NAD(P)H Dehydrogenase (Quinone)