Neonatal tremor episodes and hyperekplexia-like presentation at onset in a child with SCN8A developmental and epileptic encephalopathy

Epileptic Disord. 2018 Aug 1;20(4):289-294. doi: 10.1684/epd.2018.0988.

Abstract

SCN8A encephalopathy is a newly defined epileptic encephalopathy caused by de novo mutations of the SCN8A gene. We report herein a four-year-old boy presenting with severe non-epileptic abnormal movements, of possibly antenatal onset, progressively associated with pharmacoresistant epilepsy and regression, associated with a de novo heterozygous missense mutation of SCN8A. This case shows that paroxysmal non-epileptic episodes of severe tremor and hyperekplexia-like startles and a striking vegetative component can be the first early symptoms of severe SCN8A developmental and epileptic encephalopathy. Clinicians should be aware of these symptoms in order to avoid misdiagnosis and ensure early appropriate therapeutic management. [Published with video sequences on www.epilepticdisorders.com].

Keywords: SCN8A; developmental and epileptic encephalopathy; hyperekplexia-like; movement disorder; tremor.

Publication types

  • Case Reports

MeSH terms

  • Brain Diseases* / diagnosis
  • Brain Diseases* / genetics
  • Brain Diseases* / physiopathology
  • Child, Preschool
  • Epilepsy* / diagnosis
  • Epilepsy* / genetics
  • Epilepsy* / physiopathology
  • Humans
  • Hyperekplexia* / diagnosis
  • Hyperekplexia* / genetics
  • Hyperekplexia* / physiopathology
  • Infant, Newborn
  • Infant, Newborn, Diseases* / diagnosis
  • Infant, Newborn, Diseases* / genetics
  • Infant, Newborn, Diseases* / physiopathology
  • Male
  • NAV1.6 Voltage-Gated Sodium Channel / genetics*
  • Tremor* / diagnosis
  • Tremor* / genetics
  • Tremor* / physiopathology

Substances

  • NAV1.6 Voltage-Gated Sodium Channel
  • SCN8A protein, human