Cognitive reserve maps the core loci of neurodegeneration in corticobasal degeneration

Eur J Neurol. 2018 Nov;25(11):1333-1340. doi: 10.1111/ene.13729. Epub 2018 Jul 21.

Abstract

Background and purpose: Cognitively stimulating life experiences and activities are deemed to moderate the clinical impact of brain damage progressively building a neural and cognitive reserve (CR). CR has been studied extensively in various neurodegenerative disorders, but not in corticobasal degeneration (CBD).

Methods: Using Statistical Parametric Mapping 8, years of education, as a determinant of CR, was correlated with tracer uptake on positron emission tomography with 18 F-fluorodeoxyglucose, as a marker of neurodegeneration, in 35 patients with various phenotypes of CBD, including a cognitive-motor composite score or symptoms duration as covariates for controlling disease stage.

Results: A cluster of relative hypometabolism was found associated with higher education in the left inferior regions of pre- and post-rolandic gyri and insula, which represent typical loci of neurodegeneration in CBD regardless of clinical presentation.

Conclusions: The present findings extend to CBD the evidence gathered in other neurodegenerative disorders that a higher CR has a protective effect against the clinical manifestations of brain degeneration.

Keywords: FDG positron emission tomography; cognitive reserve; corticobasal degeneration.

MeSH terms

  • Aged
  • Aged, 80 and over
  • Basal Ganglia Diseases / diagnostic imaging*
  • Basal Ganglia Diseases / psychology
  • Cognitive Reserve / physiology*
  • Female
  • Fluorodeoxyglucose F18
  • Humans
  • Male
  • Nerve Degeneration / diagnostic imaging*
  • Nerve Degeneration / psychology
  • Positron-Emission Tomography / methods

Substances

  • Fluorodeoxyglucose F18