Biological alterations in the primary affective disorders and other tricyclic-responsive disorders

Prog Neuropsychopharmacol Biol Psychiatry. 1985;9(1):15-24. doi: 10.1016/0278-5846(85)90175-7.

Abstract

Noradrenergic function was studied in patients with primary affective disorder and other tricyclic-responsive disorders including obsessive-compulsive disorder, anorexia nervosa and panic attack/agoraphobia in medication-free states. Pre-synaptic noradrenergic activity was assessed by assaying plasma concentrations of norepinephrine (NE) and its metabolite 3-methoxy,4-hydroxyphenylglycol (MHPG). Noradrenergic receptor responsiveness was evaluated by measuring plasma growth hormone (GH), MHPG, and NE responses to clonidine. Binding of tritiated dihydroergocriptine (3H-DHE) and biochemical responsiveness of alpha 2-adrenergic receptors were measured in platelet preparations. These studies suggest that noradrenergic activity may be altered in several tricyclic-responsive disorders and are consistent with the possibility that tricyclic antidepressants may serve to stabilize a dysregulated noradrenergic system in patients from several diagnostic categories.

MeSH terms

  • Adult
  • Agoraphobia / physiopathology
  • Anorexia Nervosa / physiopathology
  • Clonidine / pharmacology*
  • Cyclic AMP / metabolism
  • Depressive Disorder / drug therapy
  • Depressive Disorder / physiopathology*
  • Dihydroergotoxine / metabolism
  • Growth Hormone / blood
  • Humans
  • Mental Disorders / blood
  • Mental Disorders / drug therapy
  • Mental Disorders / physiopathology*
  • Methoxyhydroxyphenylglycol / blood
  • Middle Aged
  • Norepinephrine / blood
  • Obsessive-Compulsive Disorder / physiopathology
  • Panic / physiology
  • Receptors, Adrenergic / physiology*
  • Tritium

Substances

  • Receptors, Adrenergic
  • Tritium
  • Dihydroergotoxine
  • Methoxyhydroxyphenylglycol
  • Growth Hormone
  • Cyclic AMP
  • Clonidine
  • Norepinephrine