Concurrent miR-21 suppression and FXR activation as a mechanism of improvement in nonalcoholic fatty liver disease

Cell Death Dis. 2018 Mar 2;9(3):354. doi: 10.1038/s41419-018-0386-3.

Authors

Guilherme S Mazzini^{1

2}, Jad Khoraki³, Matthew G Browning³, Guilherme M Campos³

Affiliations

¹ Department of Gastrointestinal Surgery, Hospital de Clinicas de Porto Alegre, 2350 Ramiro Bracelos Street, Porto Alegre, RS, Brazil. guilherme.mazzini@vcuhealth.org.
² Department of Surgery, Virginia Commonwealth University, 1200 East Broad Street, Richmond, VA, USA. guilherme.mazzini@vcuhealth.org.
³ Department of Surgery, Virginia Commonwealth University, 1200 East Broad Street, Richmond, VA, USA.

No abstract available

Publication types

Research Support, Non-U.S. Gov't
Comment

MeSH terms

Animals
Chenodeoxycholic Acid / analogs & derivatives
Liver
Mice
MicroRNAs*
Non-alcoholic Fatty Liver Disease*

Substances

MicroRNAs
obeticholic acid
Chenodeoxycholic Acid