TRPA1 channel participates in tacrolimus-induced pruritus in a chronic contact hypersensitivity murine model

J Dermatol Sci. 2018 Feb;89(2):207-209. doi: 10.1016/j.jdermsci.2017.10.012. Epub 2017 Oct 31.

¹ Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan; Department of Dermatology, Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine, Kaohsiung, Taiwan.
² Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan; Translational research department for skin and brain diseases, Kyoto University Graduate School of Medicine, 54 Shogoin-Kawara, Sakyo-ku, Kyoto, Japan. Electronic address: otsukamn@kuhp.kyoto-u.ac.jp.
³ Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan; Central Pharmaceutical Research Institute, Japan Tobacco, Japan.
⁴ Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan.
⁵ Department of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan; Singapore Immunology Network (SIgN) and Institute of Medical Biology, Agency for Science, Technology and Research (A*STAR), Biopolis, 139668, Singapore. Electronic address: kaba@kuhp.kyoto-u.ac.jp.

No abstract available

Keywords: Atopic dermatitis; Contact hypersensitivity; TRPA1 channel; Tacrolimus.

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