Clorgyline treatment differentially affects m-chlorophenylpiperazine-induced neuroendocrine changes

Eur J Pharmacol. 1988 Jun 10;150(3):239-46. doi: 10.1016/0014-2999(88)90004-0.

Abstract

Intravenous administration of the 5-HT1B agonist, m-chlorophenylpiperazine (m-CPP) to rats produced increases in plasma prolactin (peak effect at 15 min), corticosterone (peak effect at 30 min) and a decrease in plasma growth hormone (peak effect at 15 min) concentrations. Short-term or long-term clorgyline treatment did not affect baseline levels of prolactin, corticosterone or growth hormone. Short-term clorgyline treatment attenuated m-CPP's effect on corticosterone but not on prolactin or growth hormone. On the other hand, long-term clorgyline treatment attenuated m-CPP's effect on prolactin but not on corticosterone or growth hormone. These findings are compatible with development of functional subsensitivity of 5-HT1B receptors mediating prolactin release following long-term clorgyline treatment. Attenuation of m-CPP's effect on corticosterone following short-term clorgyline treatment suggests either early adaptational changes in a 5-HT receptor subtype mediating corticosterone release, or clorgyline-induced increases in other neurotransmitters such as norepinephrine which may be responsible for attenuating m-CPP's effect on corticosterone.

MeSH terms

  • Adrenal Cortex / drug effects
  • Adrenal Cortex / metabolism
  • Animals
  • Clorgyline / pharmacology*
  • Corticosterone / blood*
  • Growth Hormone / blood
  • Injections, Intravenous
  • Male
  • Neurosecretory Systems / drug effects*
  • Neurotransmitter Agents / metabolism
  • Piperazines / pharmacology*
  • Prolactin / blood*
  • Propylamines / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Receptors, Serotonin / drug effects

Substances

  • Neurotransmitter Agents
  • Piperazines
  • Propylamines
  • Receptors, Serotonin
  • Prolactin
  • Growth Hormone
  • Clorgyline
  • 1-(3-chlorophenyl)piperazine
  • Corticosterone