Regulation of immune and neural function via leukocyte Ig-like receptors

J Biochem. 2017 Aug 1;162(2):73-80. doi: 10.1093/jb/mvx036.

Abstract

Leukocyte Ig-like receptors (LILRs)/Ig-like transcripts (ILTs) are expressed on innate and adaptive immune cells and maintain immune homeostasis. LILRs consist of activating and inhibitory-type receptors that regulate adequate cellular functions. LILRs were firstly identified as MHC class I receptors, therefore expression and/or polymorphisms of LILRs are reported to associate with autoimmune disorders and transplant rejection; however, recent accumulating evidences have revealed that LILRs recognize with diverse ligands including bacteria and virus. In addition, inhibitory LILRB2 (ILT4) and murine relative paired Ig-like receptor (PIR)-B are expressed on neuron and is involved in the dysregulation of central nervous system via interaction with neuronal ligands including amyloid β-protein. In this review, we summarize recent discoveries on the functions of inhibitory MHC class I receptors, and discuss their regulatory roles in immune responses and neural functions.

Keywords: MHC class I; Nogo; amyloid β; leukocyte Ig-like receptors; paired Ig-like receptor.

Publication types

  • Review

MeSH terms

  • Animals
  • Humans
  • Leukocytes / immunology*
  • Neurons / immunology*
  • Receptors, Immunologic / immunology*

Substances

  • Receptors, Immunologic