Excitatory amino acid signal transduction in cerebellar cell cultures

Funct Neurol. 1986 Oct-Dec;1(4):345-9.

Abstract

In primary cultures of cerebellar granule cells excitatory amino acid recognition sites are coupled with the stimulation of inositol phospholipid (PI) hydrolysis, cGMP formation and 45Ca2+ uptake. Mg2+, 2-amino-5-phosphonovalerate (APV) and phencyclidine (PCP) potently inhibit signal transduction in response to N-methyl-D-aspartate (NMDA), glutamate (GLU) and aspartate (ASP). Activation by quisqualate (QUIS) is transduced selectively into stimulation of PI hydrolysis and this response is not sensitive to inhibition by Mg2+, APV and PCP. Activation by kainate (KA) is transduced into potent stimulation of cGMP formation and 45Ca2+ uptake. Transduction of KA signal is not affected by Mg2+ and is relatively insensitive to PCP and APV.

MeSH terms

  • Amino Acids / antagonists & inhibitors
  • Amino Acids / pharmacology*
  • Animals
  • Aspartic Acid / analogs & derivatives
  • Aspartic Acid / pharmacology
  • Calcium / metabolism*
  • Cells, Cultured
  • Cerebellum / cytology
  • Cerebellum / drug effects
  • Cerebellum / metabolism*
  • Cyclic GMP / metabolism*
  • Glutamates / pharmacology
  • Glutamic Acid
  • Kainic Acid / pharmacology
  • N-Methylaspartate
  • Oxadiazoles / pharmacology
  • Phosphatidylinositols / metabolism*
  • Quisqualic Acid
  • Rats

Substances

  • Amino Acids
  • Glutamates
  • Oxadiazoles
  • Phosphatidylinositols
  • Aspartic Acid
  • Glutamic Acid
  • N-Methylaspartate
  • Quisqualic Acid
  • Cyclic GMP
  • Kainic Acid
  • Calcium