Immunosenescence Induced by Plasma from Individuals with Obesity Caused Cell Signaling Dysfunction and Inflammation

Obesity (Silver Spring). 2017 Sep;25(9):1523-1531. doi: 10.1002/oby.21888. Epub 2017 Jul 14.

Abstract

Objective: To evaluate the consequences of plasma from individuals with obesity on parameters associated with immunosenescence in unrelated healthy peripheral blood mononuclear cells (PBMC).

Methods: Freshly isolated PBMC were incubated in media supplemented with 10% of plasma from individuals with obesity or control subjects for the first 4 hours of 24 to 120 hours of culture.

Results: Plasma from individuals with obesity modulated the phenotype of healthy PBMC, leading to a higher rate of apoptosis, lower amounts of phospho-γH2AX and -p53, and mitochondrial dysfunction. After 120 hours, there was a higher secretion of inflammatory cytokines IL-1β and IL-8. CD8+ T lymphocytes presented decreased expression of CD28, which is associated with the immunosenescent phenotype. CD14+ macrophages showed increased expression of CD80 and CD206, suggesting a modulation in the activation of macrophages.

Conclusions: These results demonstrate that chronic systemic inflammation observed in obesity induces dysfunctional features in PBMC that are consistent with premature immunosenescence.

MeSH terms

  • Adult
  • Apoptosis
  • CD8-Positive T-Lymphocytes / physiology
  • Culture Media
  • Female
  • Humans
  • Immunosenescence*
  • Inflammation / etiology*
  • Interleukin-1beta / metabolism
  • Interleukin-8 / metabolism
  • Leukocytes, Mononuclear / physiology*
  • Macrophages
  • Male
  • Obesity / blood*
  • Serum
  • Signal Transduction / physiology*

Substances

  • Culture Media
  • Interleukin-1beta
  • Interleukin-8