Endogenous opioids in cerebrospinal fluid of opioid-dependent humans

Biol Psychiatry. 1988 Oct;24(6):649-62. doi: 10.1016/0006-3223(88)90139-4.

Abstract

Endogenous opioid systems may be altered as a consequence of addiction, but evidence to support this idea is meager so far. We obtained 136 cerebrospinal fluid (CSF) samples from 72 opioid addicts during four distinct states: methadone maintenance, detoxification from methadone, opioid antagonist treatment, and drug-free status. CSF endorphins were measured in 86 patients samples using a radioreceptor assay (RRA), and beta-endorphin levels were measured in 85 patient samples using a radioimmuno assay (RIA). During detoxification, both RRA fraction I and beta-endorphin showed a generally similar pattern of changes. Both were lowest when measured 40-50 hr after the last opioid dose, and both showed an apparent rebound to higher than methadone maintenance values at 60-70 hr following the last dose. During methadone maintenance and drug-free states, the addicts' levels of fraction I RRA endorphins in the CSF were higher than levels found in a normal control group. Fraction II endorphins were also elevated in the addicts who were drug free. In contrast, CSF beta-endorphin during both methadone maintenance and drug-free states was lower in the addicts as compared to the normal, drug-naive group. Except for the pattern found during detoxification, there were no consistent changes in endorphin levels across different states of addiction.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Heroin Dependence / cerebrospinal fluid*
  • Heroin Dependence / rehabilitation
  • Humans
  • Male
  • Methadone / therapeutic use
  • Naltrexone / therapeutic use
  • Radioimmunoassay
  • Radioligand Assay
  • Receptors, Opioid / metabolism*
  • Substance Withdrawal Syndrome / cerebrospinal fluid
  • beta-Endorphin / cerebrospinal fluid*

Substances

  • Receptors, Opioid
  • Naltrexone
  • beta-Endorphin
  • Methadone