Abstract
Various mechanisms of resistance to ALK inhibitors in ALK rearranged adenocarcinomas are reported including secondary gatekeeper mutations, bypass signaling or ALK amplifications as the commonest ones. We report a new mechanism of resistance with transformation of adenocarcinoma into small cell lung cancer. A 53-year-old non-smoker woman, experienced cancer progression after two lines of ALK inhibitors. A new biopsy showed a small cell lung cancer. FISH revealed persistent ALK rearrangement with an atypical pattern and complete loss of the 5' region of the ALK gene.
Keywords:
ALK translocation; Lung neoplasms; Small cell cancer transformation.
Copyright © 2016 Elsevier Ireland Ltd. All rights reserved.
MeSH terms
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Adenocarcinoma / drug therapy*
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Adenocarcinoma / enzymology
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Adenocarcinoma / genetics*
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Adenocarcinoma / pathology
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Adenocarcinoma of Lung
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Anaplastic Lymphoma Kinase
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Crizotinib
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Drug Resistance, Neoplasm
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Female
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Gene Rearrangement
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Humans
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Lung Neoplasms / drug therapy*
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Lung Neoplasms / enzymology
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Lung Neoplasms / genetics*
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Lung Neoplasms / pathology
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Middle Aged
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Mutation
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Positron Emission Tomography Computed Tomography
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Protein Kinase Inhibitors / pharmacology
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Protein Kinase Inhibitors / therapeutic use*
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Pyrazoles / pharmacology
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Pyrazoles / therapeutic use*
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Pyridines / pharmacology
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Pyridines / therapeutic use*
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Receptor Protein-Tyrosine Kinases / antagonists & inhibitors*
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Receptor Protein-Tyrosine Kinases / genetics*
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Tomography Scanners, X-Ray Computed
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Translocation, Genetic
Substances
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Protein Kinase Inhibitors
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Pyrazoles
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Pyridines
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Crizotinib
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ALK protein, human
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Anaplastic Lymphoma Kinase
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Receptor Protein-Tyrosine Kinases