Consumption of Dietary Resistant Starch Partially Corrected the Growth Pattern Despite Hyperglycemia and Compromised Kidney Function in Streptozotocin-Induced Diabetic Rats

J Agric Food Chem. 2016 Oct 12;64(40):7540-7545. doi: 10.1021/acs.jafc.6b03808. Epub 2016 Sep 30.

Abstract

We previously demonstrated that feeding of dietary resistant starch (RS) prior to the induction of diabetes delayed the progression of diabetic nephropathy and maintained vitamin D balance in streptozotocin (STZ)-induced type 1 diabetic (T1D) rats. Here, we examined the impact of RS on kidney function and vitamin D homeostasis following STZ injection. Male Sprague-Dawley rats were administered STZ and fed a standard diet containing cornstarch or 20, 10, or 5% RS for 4 weeks. T1D rats fed 10 and 20% RS, but not 5% RS, gained more weight than cornstarch-fed rats. Yet, renal health and glucose metabolism were not improved by RS. Our data suggest that RS normalized growth patterns in T1D rats after diabetes induction in a dose-dependent manner despite having no effect on blood glucose and vitamin D balances. Future interventions should focus on the preventative strategies with RS in T1D.

Keywords: 25-hydroxycholecalciferol; diabetic nephropathy; high-amylose maize; resistant starch; streptozotocin; type 1 diabetes; vitamin D.

MeSH terms

  • Animals
  • Body Weight / drug effects
  • Diabetes Mellitus, Experimental / diet therapy*
  • Diabetes Mellitus, Experimental / physiopathology
  • Diabetes Mellitus, Type 1 / diet therapy
  • Hyperglycemia / metabolism
  • Interleukin-6 / blood
  • Kidney / drug effects*
  • Kidney / physiology
  • Kidney Function Tests
  • Male
  • Rats, Sprague-Dawley
  • Starch / chemistry
  • Starch / pharmacology*
  • Streptozocin
  • Tumor Necrosis Factor-alpha / blood
  • Vitamin D / blood

Substances

  • Interleukin-6
  • Tumor Necrosis Factor-alpha
  • Vitamin D
  • Streptozocin
  • Starch