Resistance to drugs associated with the multidrug resistance phenotype following selection with high-concentration methotrexate

J Natl Cancer Inst. 1989 Aug 16;81(16):1250-4. doi: 10.1093/jnci/81.16.1250.

Abstract

To study patterns of resistance at extreme but nevertheless clinically relevant drug concentrations, we developed a series of methotrexate-selected CCRF-CEM sublines, all of which were highly resistant to this antifolate (relative resistance, 10(2)- to greater than 10(5)-fold). The least methotrexate-resistant subline was completely sensitive to drugs associated with the multidrug resistance phenotype. However, more highly methotrexate-resistant sublines were significantly cross-resistant to vincristine, vinblastine, and dactinomycin (maximum relative resistance, 40-fold). These sublines were not cross-resistant to doxorubicin, daunorubicin, and teniposide. Regression analysis indicated that relative resistance to methotrexate was correlated with relative resistance to vincristine (r = 0.96) and vinblastine (r = 0.99). Such cross-resistance in highly methotrexate-resistant cells may have important clinical implications.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Cell Survival / drug effects
  • Dactinomycin / pharmacology
  • Drug Resistance
  • Drug Screening Assays, Antitumor
  • Humans
  • Leukemia / genetics
  • Leukemia / pathology*
  • Methotrexate / administration & dosage
  • Methotrexate / pharmacology*
  • Phenotype
  • Regression Analysis
  • Tumor Cells, Cultured / drug effects
  • Tumor Cells, Cultured / pathology
  • Vinblastine / pharmacology
  • Vincristine / pharmacology

Substances

  • Antineoplastic Agents
  • Dactinomycin
  • Vincristine
  • Vinblastine
  • Methotrexate