Protective effect of chlorpromazine against the lethality of interleukin 1 in adrenalectomized or actinomycin D-sensitized mice

Biochem Biophys Res Commun. 1989 Dec 29;165(3):942-6. doi: 10.1016/0006-291x(89)92694-6.

Abstract

Interleukin 1 (IL-1) and Tumor Necrosis Factor (TNF) are thought to play a key role in septic shock and inflammation. We have tested the effect of dexamethasone (DEX) and chlorpromazine (CPZ) on the lethal effect of IL-1, TNF and endotoxin. Two different experimental models were used to sensitize mice to the lethal effect of IL-1: adrenalectomy and pretreatment with actinomycin D. CPZ (4 mg/kg) was found to protect mice against IL-1 and endotoxin toxicity in all cases, while DEX had a protective effect only in adrenalectomized mice. In contrast to its protective effect against IL-1 and endotoxin, CPZ did not protect mice against TNF. These findings might be useful in the analysis of the differences in the actions of IL-1 and TNF in vivo, and in the development of new drugs preventing their toxicity.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adrenalectomy*
  • Animals
  • Chlorpromazine / pharmacology*
  • Dactinomycin / pharmacology*
  • Dexamethasone / pharmacology
  • Escherichia coli
  • Interleukin-1 / toxicity*
  • Lipopolysaccharides / pharmacology
  • Male
  • Mice
  • Tumor Necrosis Factor-alpha / toxicity

Substances

  • Interleukin-1
  • Lipopolysaccharides
  • Tumor Necrosis Factor-alpha
  • Dactinomycin
  • Dexamethasone
  • Chlorpromazine