Control of APC/C-dependent ubiquitin chain elongation by reversible phosphorylation

Proc Natl Acad Sci U S A. 2016 Feb 9;113(6):1540-5. doi: 10.1073/pnas.1522423113. Epub 2016 Jan 25.

Abstract

Most metazoan E3 ligases contain a signature RING domain that promotes the transfer of ubiquitin from the active site of E2 conjugating enzymes to lysine residues in substrates. Although these RING-E3s depend on E2 enzymes for catalysis, how they turn on their E2s at the right time and place remains poorly understood. Here we report a phosphorylation-dependent mechanism that ensures timely activation of the E2 Ube2S by its RING-E3, the anaphase-promoting complex (APC/C); while phosphorylation of a specific serine residue in the APC/C coactivator Cdc20 prevents delivery of Ube2S to the APC/C, removal of this mark by PP2A(B56) allows Ube2S to bind the APC/C and catalyze ubiquitin chain elongation. PP2A(B56) also stabilizes kinetochore-microtubule attachments to shut off the spindle checkpoint, suggesting that cells regulate the E2-E3 interplay to coordinate ubiquitination with critical events during cell division.

Keywords: APC/C; Ube2S; anaphase-promoting complex; phosphorylation; ubiquitin.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anaphase-Promoting Complex-Cyclosome / metabolism*
  • Biocatalysis
  • Cdc20 Proteins / metabolism
  • HeLa Cells
  • Humans
  • Kinetochores / metabolism
  • Phosphorylation
  • Protein Binding
  • Protein Phosphatase 2 / metabolism
  • Serine / metabolism
  • Ubiquitin / metabolism*

Substances

  • Cdc20 Proteins
  • Ubiquitin
  • Serine
  • Anaphase-Promoting Complex-Cyclosome
  • Protein Phosphatase 2