Leukocyte production of IFN-γ and TNF-α in 8- to 12-y-old children with low serum iron levels

Nutrition. 2016 May;32(5):546-52. doi: 10.1016/j.nut.2015.11.005. Epub 2015 Dec 10.

Abstract

Objective: Iron deficiency is likely the most common nutritional deficiency worldwide; low iron concentrations have been related to alterations in immune system functions; therefore, the aim of this study was to determine the effect of low serum iron (LSI) concentrations on the production of proinflammatory cytokines by peripheral blood leukocytes in 8- to 12-y-old children from a local community.

Methods: We obtained 120 blood samples and determined full blood counts and serum iron concentrations. An LSI and a control group, paired by age and sex were established using serum iron <60 μg/dL as the cutoff point. Ferritin and C-reactive protein concentrations were quantified. Serum interferon (IFN)-γ and tumor necrosis factor (TNF)-α concentrations were measured in these groups by enzyme-linked immunosorbent assay. A second blood sample was taken from children in both groups to isolate peripheral blood mononuclear cells (PBMCs) and measure IFN-γ and TNF-α production by unstimulated and lipopolysaccharide/phorbol myristate acetate/ionomycin-stimulated leukocytes in vitro.

Results: Of the participants in the present study, 17.5% (21 children) presented LSI, as well as decreased ferritin concentrations. Differential counts from total blood samples showed a significant increase in leukocyte numbers in the LSI group, along with increased neutrophil frequencies and numbers but decreased lymphocyte frequencies. Decreased serum IFN-γ concentrations and decreased in vitro production of IFN-γ by PBMCs were found in the LSI group.

Conclusions: The results of the present study suggest that low iron levels alter leukocyte subpopulations in circulation and have a detrimental effect on leukocyte production of proinflammatory cytokines after an antigenic challenge.

Keywords: Cytokines; Immune response; Leukocytes; Low serum iron.

MeSH terms

  • Anemia, Iron-Deficiency / blood
  • Anemia, Iron-Deficiency / immunology
  • Anemia, Iron-Deficiency / metabolism
  • Anemia, Iron-Deficiency / physiopathology*
  • Asymptomatic Diseases*
  • Blood Cell Count
  • C-Reactive Protein / analysis
  • Calcium Ionophores / pharmacology
  • Child
  • Child Nutritional Physiological Phenomena*
  • Female
  • Ferritins / blood
  • Humans
  • Interferon-gamma / biosynthesis
  • Interferon-gamma / blood
  • Interferon-gamma / metabolism*
  • Ionomycin / pharmacology
  • Iron / blood
  • Leukocytes, Mononuclear / drug effects
  • Leukocytes, Mononuclear / immunology*
  • Leukocytes, Mononuclear / metabolism
  • Leukocytosis / etiology*
  • Lipopolysaccharides / pharmacology
  • Male
  • Mexico
  • Mitogens / pharmacology
  • Tetradecanoylphorbol Acetate / analogs & derivatives
  • Tetradecanoylphorbol Acetate / pharmacology
  • Tumor Necrosis Factor-alpha / biosynthesis
  • Tumor Necrosis Factor-alpha / blood
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • Calcium Ionophores
  • IFNG protein, human
  • Lipopolysaccharides
  • Mitogens
  • TNF protein, human
  • Tumor Necrosis Factor-alpha
  • Ionomycin
  • phorbolol myristate acetate
  • Interferon-gamma
  • C-Reactive Protein
  • Ferritins
  • Iron
  • Tetradecanoylphorbol Acetate