Low bone mineral density in achondroplasia and hypochondroplasia

Pediatr Int. 2016 Aug;58(8):705-8. doi: 10.1111/ped.12890. Epub 2016 Apr 5.

Abstract

Background: Achondroplasia (ACH) and hypochondroplasia (HCH) are the most common form of short-limb skeletal dysplasias caused by activated fibroblast growth factor receptor 3 (FGFR3) signaling. Although decreased bone mass was reported in gain-of-function mutation in Fgfr3 mice, both disorders have never been described as osteoporotic. In the present study, we evaluated bone mineral density (BMD) in ACH and HCH patients.

Methods: We measured spinal BMD (L1-L4) in 18 ACH and four HCH patients with an average age of 19.8 ± 7.5 years (range, 10-33 years). BMD Z-score in each individual was calculated for normalizing age and gender. Correlation between body mass index (BMI) and BMD was analyzed. Moreover, BMD and Z-score were compared between ACH patients and HCH patients.

Results: The average BMD of ACH/HCH patients was 0.805 ± 0.141 g/cm(2) (range, 0.554-1.056 g/cm(2) ), resulting in an average Z-score of -1.1 ± 0.8 (range, -2.4 to 0.6) of the standard value. A slightly positive correlation was observed between BMI and BMD (r = 0.45; P = 0.13). There was no significant difference in BMD and Z-score between ACH and HCH patients.

Conclusion: Spinal BMD was reduced in ACH/HCH patients, and was mildly correlated with individual BMI. We should carefully monitor BMD and examine osteoporosis-related symptoms in adolescent and adult ACH/HCH patients. © 2016 Japan Pediatric Society.

Keywords: achondroplasia; bone mineral density; fibroblast growth factor receptor 3; hypochondroplasia; osteoporosis.

MeSH terms

  • Absorptiometry, Photon
  • Achondroplasia / diagnosis*
  • Achondroplasia / genetics
  • Achondroplasia / metabolism
  • Adolescent
  • Adult
  • Bone Density / physiology*
  • Bone and Bones / abnormalities*
  • Bone and Bones / metabolism
  • Child
  • DNA Mutational Analysis
  • Dwarfism / diagnosis*
  • Dwarfism / genetics
  • Dwarfism / metabolism
  • Female
  • Humans
  • Japan
  • Limb Deformities, Congenital / diagnosis*
  • Limb Deformities, Congenital / genetics
  • Limb Deformities, Congenital / metabolism
  • Lordosis / diagnosis*
  • Lordosis / genetics
  • Lordosis / metabolism
  • Lumbar Vertebrae / diagnostic imaging
  • Male
  • Mutation
  • Receptor, Fibroblast Growth Factor, Type 3 / genetics
  • Receptor, Fibroblast Growth Factor, Type 3 / metabolism
  • Young Adult

Substances

  • FGFR3 protein, human
  • Receptor, Fibroblast Growth Factor, Type 3

Supplementary concepts

  • Hypochondroplasia