A nanomedicine based combination therapy based on QLPVM peptide functionalized liposomal tamoxifen and doxorubicin against Luminal A breast cancer

Nanomedicine. 2016 Feb;12(2):387-97. doi: 10.1016/j.nano.2015.12.360. Epub 2015 Dec 19.

Abstract

Though combination chemotherapy or antitumor nanomedicine is extensively investigated, their combining remains in infancy. Additionally, enhanced delivery of estrogen or its analogs to tumor with highly-expressed estrogen-receptor (ER) is seldom considered, despite its necessity for ER-positive breast cancer treatment. Here, nanomedicine based combination therapy using QLPVM conjugated liposomal tamoxifen (TAM) and doxorubicin (DOX) was designed and testified, where the penta-peptide was derived from Ku70 Bax-binding domain. Quantitative, semi-quantitative and qualitative approaches demonstrated the enhanced endocytosis and cytotoxicity of QLPVM conjugated sterically stabilized liposomes (QLPVM-SSLs) in vitro and in vivo. Mechanism studies of QLPVM excluded the possible electrostatic, hydrophobic or receptor-ligand interactions. However, as a weak cell-penetrating peptide, QLPVM significantly induced drug release from QLPVM-SSLs during their interaction with cells, which was favorable for drug internalization. These findings suggested that the nanomedicine based combination therapy using QLPVM-SSL-TAM and QLPVM-SSL-DOX might provide a rational strategy for Luminal A breast cancer.

From the clinical editor: Breast cancer remains a leading cause of mortality in women worldwide. Although combined therapy using hormonal antagonist and chemotherapy is the norm nowadays, the use of these agents together in a single delivery system has not been tested. Here, the authors investigated this approach using QLPVM conjugated liposomes in in-vitro and in-vivo models. The positive findings may provide a novel direction for breast cancer treatment in the near future.

Keywords: Doxorubicin; Luminal A breast cancer; Nanomedicine based combinational therapy; Penta-peptide QLPVM; Tamoxifen.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibiotics, Antineoplastic / administration & dosage
  • Antibiotics, Antineoplastic / therapeutic use
  • Antineoplastic Agents, Hormonal / administration & dosage
  • Antineoplastic Agents, Hormonal / therapeutic use
  • Antineoplastic Combined Chemotherapy Protocols / administration & dosage*
  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use
  • Breast / drug effects
  • Breast / metabolism
  • Breast / pathology
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / pathology
  • Cell Line, Tumor
  • Cell-Penetrating Peptides / chemistry
  • Cell-Penetrating Peptides / metabolism*
  • Doxorubicin / administration & dosage
  • Doxorubicin / analogs & derivatives*
  • Doxorubicin / therapeutic use
  • Drug Combinations
  • Female
  • Humans
  • Ku Autoantigen / chemistry
  • Ku Autoantigen / metabolism
  • Liposomes / chemistry
  • Liposomes / metabolism*
  • Mice, Nude
  • Nanomedicine
  • Oligopeptides / chemistry
  • Oligopeptides / metabolism*
  • Polyethylene Glycols / administration & dosage
  • Polyethylene Glycols / therapeutic use
  • Tamoxifen / administration & dosage*
  • Tamoxifen / therapeutic use
  • bcl-2-Associated X Protein / metabolism

Substances

  • Antibiotics, Antineoplastic
  • Antineoplastic Agents, Hormonal
  • Cell-Penetrating Peptides
  • Drug Combinations
  • Liposomes
  • Oligopeptides
  • bcl-2-Associated X Protein
  • liposomal doxorubicin
  • Tamoxifen
  • Polyethylene Glycols
  • Doxorubicin
  • Ku Autoantigen