Abstract
In this issue of Cancer Cell, Giachino and colleagues, employing various approaches, describe a tumor suppressor function for Notch signaling in forebrain tumors and suggest that decreased Notch activity could be a key molecular event in supratentorial primitive neuroectodermal tumors (sPNET).
Copyright © 2015 Elsevier Inc. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
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Comment
MeSH terms
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Animals
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Brain Neoplasms / metabolism*
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Glioma / metabolism*
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Humans
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Neoplastic Stem Cells / metabolism*
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Neural Stem Cells / metabolism*
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Prosencephalon / metabolism*
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Receptors, Notch / metabolism*
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Signal Transduction*
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Tumor Suppressor Proteins / metabolism*
Substances
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Receptors, Notch
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Tumor Suppressor Proteins