Association between NAD(P)H oxidase p22phox gene variants and acute myocardial infarction in a Han Chinese population

Herz. 2016 Aug;41(5):428-34. doi: 10.1007/s00059-015-4391-3. Epub 2015 Dec 11.

Abstract

Background: We investigated the association between five selected single-nucleotide polymorphisms (rs12933505, rs3180279, rs3794264, rs4673, rs1049255) in the NAD(P)H p22phox gene and acute myocardial infarction (AMI) as well as severity of coronary artery stenosis in a Han Chinese population.

Patients and methods: A total of 168 patients with AMI and 138 healthy controls were recruited. The TaqMan allelic discrimination assay was used to genotype five single-nucleotide polymorphisms.

Results: The frequency of the rs1049255 G allele was significantly lower in patients with AMI than in controls (p = 0.022). Compared with subjects with an AA genotype, subjects with a GG or AG genotype had a lower risk of AMI [multivariate-adjusted odds ratio (OR), 0.53; 95 % CI, 0.29-0.95; p = 0.031). Subjects with the GG and AG genotypes of rs1049255 showed a decreased susceptibility for triple-vessel disease (TVD) as compared with controls (multivariate-adjusted OR, 0.43; 95 % CI, 0.19-0.98; p = 0.042). Multiple logistic regression analysis revealed that the rs1049255G variant was an independent protective factor for AMI/TVD.

Conclusion: The results suggest there is an association between the p22phox rs1049255 polymorphism with the prevalence of AMI and the severity of coronary artery stenosis in the Han Chinese population.

Keywords: Coronary artery disease; Gene polymorphism; Myocardial infarction; NAD(P)H oxidase; p22phox gene.

MeSH terms

  • Age Distribution
  • Biomarkers / analysis
  • China / epidemiology
  • China / ethnology
  • Coronary Stenosis / epidemiology*
  • Coronary Stenosis / genetics*
  • Female
  • Genetic Association Studies
  • Genetic Markers / genetics
  • Genetic Predisposition to Disease / epidemiology*
  • Genetic Predisposition to Disease / genetics*
  • Genetic Variation / genetics
  • Humans
  • Incidence
  • Male
  • Middle Aged
  • NADPH Oxidases / genetics*
  • Polymorphism, Single Nucleotide / genetics*
  • Reproducibility of Results
  • Risk Assessment / methods
  • Sensitivity and Specificity
  • Sex Distribution

Substances

  • Biomarkers
  • Genetic Markers
  • NADPH Oxidases
  • CYBA protein, human