Intranasal administration of poly-gamma glutamate induced antiviral activity and protective immune responses against H1N1 influenza A virus infection

Virol J. 2015 Oct 6:12:160. doi: 10.1186/s12985-015-0387-0.

Abstract

Background: The global outbreak of a novel swine-origin strain of the 2009 H1N1 influenza A virus and the sudden, worldwide increase in oseltamivir-resistant H1N1 influenza A viruses highlight the urgent need for novel antiviral therapy.

Methods: Here, we investigated the antiviral efficacy of poly-gamma glutamate (γ-PGA), a safe and edible biomaterial that is naturally synthesized by Bacillus subtilis, against A/Puerto Rico/8/1934 (PR8) and A/California/04/2009 (CA04) H1N1 influenza A virus infections in C57BL/6 mice.

Results: Intranasal administration of γ-PGA for 5 days post-infection improved survival, increased production of antiviral cytokines including interferon-beta (IFN-β) and interleukin-12 (IL-12), and enhanced activation of natural killer (NK) cells and influenza antigen-specific cytotoxic T lymphocytes (CTL) activity.

Conclusions: These results suggest that γ-PGA protects mice against H1N1 influenza A virus by enhancing antiviral immune responses.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Administration, Intranasal
  • Animals
  • Disease Models, Animal
  • Female
  • Immunologic Factors / administration & dosage*
  • Influenza A Virus, H1N1 Subtype / immunology*
  • Mice, Inbred C57BL
  • Orthomyxoviridae Infections / prevention & control*
  • Polyglutamic Acid / administration & dosage
  • Polyglutamic Acid / analogs & derivatives*
  • Survival Analysis

Substances

  • Immunologic Factors
  • poly(gamma-glutamic acid)
  • Polyglutamic Acid