Abstract
Inflammasome complexes form upon interaction of Nod Like Receptor (NLR) proteins with pathogen associated molecular patterns (PAPMS) inside the cytosol. Stimulation of a subset of inflammasome receptors including NLRP3, NLRC4 and AIM2 triggers formation of the micrometer-sized spherical supramolecular complex called the ASC speck. The ASC speck is thought to be the platform of inflammasome activity, but the reason why a supramolecular complex is preferred against oligomeric platforms remains elusive. We observed that a set of cytosolic proteins, including the model antigen ovalbumin, tend to co-aggregate on the ASC speck. We suggest that co-aggregation of antigenic proteins on the ASC speck during intracellular infection might be instrumental in antigen presentation.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antigen Presentation*
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Antigens / chemistry
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CARD Signaling Adaptor Proteins / metabolism*
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Calcium-Binding Proteins / metabolism*
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Carrier Proteins / metabolism*
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Caspase 1 / metabolism
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Cell Differentiation
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Cytoskeleton / metabolism
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Cytosol / metabolism
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DNA-Binding Proteins / metabolism*
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Green Fluorescent Proteins / metabolism
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HEK293 Cells
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Humans
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Inflammasomes / metabolism*
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Inflammation
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Macrophages / metabolism
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NLR Family, Pyrin Domain-Containing 3 Protein
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Ovalbumin / chemistry
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Proteasome Endopeptidase Complex / metabolism
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Tetradecanoylphorbol Acetate / chemistry
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Ubiquitin / metabolism
Substances
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AIM2 protein, human
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Antigens
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CARD Signaling Adaptor Proteins
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Calcium-Binding Proteins
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Carrier Proteins
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DNA-Binding Proteins
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Inflammasomes
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NLR Family, Pyrin Domain-Containing 3 Protein
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NLRC4 protein, human
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NLRP3 protein, human
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Ubiquitin
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Green Fluorescent Proteins
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Ovalbumin
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Caspase 1
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Proteasome Endopeptidase Complex
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Tetradecanoylphorbol Acetate
Grants and funding
This project was supported by the European Molecular Biology Organization Installation Grant (EMBO-SDIG 1468) and Boğaziçi University Research Fund (BAP-6526 and BAP-TUG 8400 Grants) Grant to N.Ö.