A novel AIFM1 mutation expands the phenotype to an infantile motor neuron disease

Eur J Hum Genet. 2016 Mar;24(3):463-6. doi: 10.1038/ejhg.2015.141. Epub 2015 Jul 15.

Abstract

AIFM1 is a gene located on the X chromosome, coding for AIF (Apoptosis-Inducing Factor), a mitochondrial flavoprotein involved in caspase-independent cell death. AIFM1 mutations have been associated with different clinical phenotypes: a severe infantile encephalopathy with combined oxidative phosphorylation deficiency and the Cowchock syndrome, an X-linked Charcot-Marie-Tooth disease (CMTX4) with axonal sensorimotor neuropathy, deafness and cognitive impairment. In two male cousins with early-onset mitochondrial encephalopathy and cytochrome c oxidase (COX) deficiency, we identified a novel AIFM1 mutation. Muscle biopsies and electromyography in both patients showed signs of severe denervation. Our patients manifested a phenotype that included signs of both cortical and motor neuron involvement. These observations emphasize the role of AIF in the development and function of neurons.

Publication types

  • Case Reports

MeSH terms

  • Amino Acid Sequence
  • Apoptosis Inducing Factor / chemistry
  • Apoptosis Inducing Factor / genetics*
  • Family
  • Genetic Predisposition to Disease*
  • Humans
  • Infant
  • Infant, Newborn
  • Male
  • Molecular Sequence Data
  • Motor Neuron Disease / genetics*
  • Mutation / genetics*
  • Phenotype
  • Sequence Alignment

Substances

  • AIFM1 protein, human
  • Apoptosis Inducing Factor