Lens status influences the association between CFH polymorphisms and age-related macular degeneration: findings from two population-based studies in Singapore

PLoS One. 2015 Mar 18;10(3):e0119570. doi: 10.1371/journal.pone.0119570. eCollection 2015.

Abstract

Aims: To determine the differential effects of genetic polymorphism in CFH and ARMS2 on risk of age-related macular degeneration (AMD) between phakic vs. pseudophakic/aphakic eyes.

Methods: 9,529 eyes of 4,918 participants from the Singapore Malay Eye Study and Singapore Indian Eye Study were analyzed. Participants had detailed eye examinations, including slit-lamp examinations and dilated fundus photography. AMD grading was performed according to the Wisconsin age-related maculopathy grading system. Lens status was defined. Single nucleotide polymorphisms (SNPs) rs10801555 (Y402H) within CFH and rs3750847 in ARMS2 were assessed. The main outcome measure was early AMD or any AMD.

Results: No significant associations between the CFH Y402H genotypes and early AMD were found in phakic individuals. In contrast, among pseudophakic/aphakic individuals, the CFH Y402H risk genotypes were significantly associated with higher odds of early AMD, with an OR of 1.57 (95% CI: 1.07-2.29) for GA genotype and 2.40 (95% CI: 1.25-4.61) for AA genotype, compared to those with GG genotype. There was significant interaction between pseudophakic/aphakic status and CFH Y402H variant on risk of early AMD (p = 0.037), adjusting for age, gender, and the first 5 genetic principal components. No significant interaction was found between lens status and ARMS2 rs3750847.

Conclusions: CFH genetic polymorphism and pseudophakic/aphakic status may have a potential synergistic effect on early AMD, suggesting roles for the complement system and related pathways in the pathogenesis of AMD in eyes after cataract surgery.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Aphakia / genetics
  • Asian People / genetics
  • Complement Factor H / genetics*
  • Female
  • Humans
  • Lens, Crystalline*
  • Macular Degeneration / epidemiology
  • Macular Degeneration / genetics*
  • Male
  • Middle Aged
  • Odds Ratio
  • Phakic Intraocular Lenses
  • Polymorphism, Single Nucleotide
  • Proteins / genetics*
  • Risk Factors
  • Singapore / epidemiology

Substances

  • ARMS2 protein, human
  • Proteins
  • Complement Factor H

Grants and funding

Supported by funding from the American Health Assistance Foundation (M2011068), and grants from the National Medical Research Council, Singapore (NMRC 0976/2003, STaR/0003/2008, CSA/033/2012, CG/SERI/2010), and the Biomedical Research Council, Singapore (BMRC 09/1/35/19/616 and 08/1/35/19/550). Ching-Yu Cheng is supported by an award from NMRC (CSA/033/2012). The Singapore Tissue Network and the Genome Institute of Singapore, Agency for Science, Technology and Research, Singapore provided services for tissue archival and genotyping, respectively. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.