Selective disruption of the AKAP signaling complexes

Methods Mol Biol. 2015:1294:137-50. doi: 10.1007/978-1-4939-2537-7_11.

Abstract

Synthesis of the second messenger cAMP activates a variety of signaling pathways critical for all facets of intracellular regulation. Protein kinase A (PKA) is the major cAMP-responsive effector. Where and when this enzyme is activated has profound implications on the cellular role of PKA. A-Kinase Anchoring Proteins (AKAPs) play a critical role in this process by orchestrating spatial and temporal aspects of PKA action. A popular means of evaluating the impact of these anchored signaling events is to biochemically interfere with the PKA-AKAP interface. Hence, peptide disruptors of PKA anchoring are valuable tools in the investigation of local PKA action. This article outlines the development of PKA isoform-selective disruptor peptides, documents the optimization of cell-soluble peptide derivatives, and introduces alternative cell-based approaches that interrogate other aspects of the PKA-AKAP interface.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • A Kinase Anchor Proteins / chemistry
  • A Kinase Anchor Proteins / metabolism*
  • Cyclic AMP / metabolism
  • Cyclic AMP-Dependent Protein Kinases / antagonists & inhibitors*
  • Cyclic AMP-Dependent Protein Kinases / metabolism
  • Peptide Library
  • Peptides / chemistry*
  • Peptides / metabolism
  • Protein Binding
  • Protein Isoforms / metabolism
  • Signal Transduction

Substances

  • A Kinase Anchor Proteins
  • Peptide Library
  • Peptides
  • Protein Isoforms
  • Cyclic AMP
  • Cyclic AMP-Dependent Protein Kinases