Androgen receptors, androgen-dependent proliferation, and 5 alpha-reductase activity of small-cell lung cancer cell lines

Int J Cancer. 1989 Apr 15;43(4):685-91. doi: 10.1002/ijc.2910430424.

Abstract

We investigated the influence of testosterone on binding to established small-cell lung cancer (SCLC) cell lines and were able to show specific high-affinity and low-capacity binding sites in some cell lines with a typical receptor size, using sucrose density gradient centrifugation. In addition, we demonstrated marked growth stimulation with testosterone and dehydrotestosterone using different androgen-receptor-positive small-cell lung cancer cell lines. This growth stimulation could be counteracted by the addition of anti-androgens like cyproterone acetate or flutamide. The presence of 5 alpha-reductase activity, 17 beta-hydroxysteroid-dehydrogenase and 3 alpha-hydroxysteroid-dehydrogenase activities could be demonstrated, suggesting testosterone metabolism of small-cell lung cancer cell lines.

MeSH terms

  • Androgen Antagonists / pharmacology
  • Androgens / metabolism
  • Androgens / pharmacology*
  • Carcinoma, Small Cell / metabolism*
  • Carcinoma, Small Cell / pathology
  • Cell Division / drug effects
  • Cell Line
  • Centrifugation, Density Gradient
  • Cholestenone 5 alpha-Reductase
  • Dose-Response Relationship, Drug
  • Humans
  • Lung Neoplasms / metabolism*
  • Lung Neoplasms / pathology
  • Oxidoreductases / analysis
  • Oxidoreductases / metabolism*
  • Radioligand Assay
  • Receptors, Androgen / drug effects
  • Receptors, Androgen / metabolism*
  • Thymidine / metabolism
  • Tritium
  • Tumor Cells, Cultured

Substances

  • Androgen Antagonists
  • Androgens
  • Receptors, Androgen
  • Tritium
  • Oxidoreductases
  • Cholestenone 5 alpha-Reductase
  • Thymidine