Effective inhibition of melanoma tumorigenesis and growth via a new complex vaccine based on NY-ESO-1-alum-polysaccharide-HH2

Mol Cancer. 2014 Jul 28:13:179. doi: 10.1186/1476-4598-13-179.

Abstract

Background: A safe and effective adjuvant plays an important role in the development of a vaccine. However, adjuvants licensed for administration in humans remain limited. Here, for the first time, we developed a novel combination adjuvant alum-polysaccharide-HH2 (APH) with potent immunomodulating activities, consisting of alum, polysaccharide of Escherichia coli and the synthetic cationic innate defense regulator peptide HH2.

Methods: The adjuvant effects of APH were examined using NY-ESO-1 protein-based vaccines in prophylactic and therapeutic models. We further determined the immunogenicity and anti-tumor effect of NY-ESO-1-APH (NAPH) vaccine using adoptive cellular/serum therapy in C57/B6 and nude mice. Cell-mediated and antibody-mediated immune responses were evaluated.

Results: The APH complex significantly promoted antigen uptake, maturation and cross-presentation of dendritic cells and enhanced the secretion of TNF-α, MCP-1 and IFN-γ by human peripheral blood mononuclear cells compared with individual components. Vaccination of NAPH resulted in significant tumor regression or delayed tumor progression in prophylactic and therapeutic models. In addition, passive serum/cellular therapy potently inhibited tumor growth of NY-ESO-1-B16. Mice treated with NAPH vaccine produced higher antibody titers and greater antibody-dependent/independent cellular cytotoxicity. Therefore, NAPH vaccination effectively stimulated innate immunity, and boosted both arms of the adaptive humoral and cellular immune responses to suppress tumorigenesis and growth of melanoma.

Conclusions: Our study revealed the potential application of APH complex as a novel immunomodulatory agent for vaccines against tumor refractory and growth.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibody Specificity / drug effects
  • Antigens, Neoplasm / immunology
  • Body Weight / drug effects
  • Cancer Vaccines / immunology*
  • Cancer Vaccines / pharmacology
  • Carcinogenesis / drug effects
  • Carcinogenesis / immunology
  • Carcinogenesis / pathology*
  • Cell Proliferation / drug effects
  • Chemokines / blood
  • Cross-Priming / drug effects
  • Dendritic Cells / drug effects
  • Dendritic Cells / immunology
  • Disease Models, Animal
  • Female
  • Humans
  • Immunity, Innate / drug effects
  • Leukocytes, Mononuclear / metabolism
  • Lymphocytes, Tumor-Infiltrating / drug effects
  • Lymphocytes, Tumor-Infiltrating / pathology
  • Melanoma / immunology*
  • Melanoma / pathology*
  • Mice, Inbred C57BL
  • NF-kappa B / metabolism
  • Polysaccharides / immunology*
  • Polysaccharides / pharmacology
  • Signal Transduction / drug effects
  • T-Lymphocytes, Cytotoxic / drug effects
  • T-Lymphocytes, Cytotoxic / immunology
  • Tumor Burden / drug effects
  • Vaccination

Substances

  • Antigens, Neoplasm
  • Cancer Vaccines
  • Chemokines
  • NF-kappa B
  • NY-ESO-1-APH vaccine
  • Polysaccharides